Neuro - Cerebellum Dysfunction - Impairments

Front 1

• What are 4 common cerebellar signs that fall under the term ataxia?
  
• What are 7 other possible Cb impairments?

Back 1

• Dysmetria, DDK, rebound/"lack of check", ataxic gait
• Hypotonia, weakness, dyssynergia/decomposition
• Tremor, nystagmus, dysarthria
• Gait/balance abnormalities

Front 2

• Why might PW-Cb dysfunction be hypotonic?

• Where might this hypotonia occur?
• How does this affect maximum force output? Sustained force output?

Back 2

• Due to the diminished cortical motor signals from the Cb

• Anywhere, but postural extensors in particular
• Max. output unaffected; Sustained force diminishes

Front 3

• How might a PW-Cb dysfunction present dysarthric? Why?
  
• What are 2 reasons a PW-Cb dysfunction may present weak?

Back 3

• Poor coordination of speech apparatus and breath support due to muscle dyssynergy results in scanning speech (pausing after syllables as if they were full words)
• Lack of timing of muscle contraction and lack of consistent direction of force production

Front 4

• Lesion of which lobe may result in nystagmus?
  
• Define dyssynergia (decomposition?) of movement

Back 4

• Flocculonodular lobe
  
• Breaking down multi-joint, multisegmental motions that result in a smooth distal segment into component pieces (instead of punching, it is shoulder flexion, then elbow extension)

Front 5

• What does the typical gait resemble in a PW-Cb dysfunction?
  
• Lesions where in the Cb may lead to this?
• How does walking with eyes closed affect the ataxia?

Back 5

• An intoxicated person's gait pattern with frequent staggering
  
• Mid-line lesions
• It would have minimal effect since visual input has little influence over cerebellar ataxia