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73 Cards in this Set

  • Front
  • Back
Types of neurotransmitters
ACh
Catecholamines
Serotonin
GABA
Glutamate
Endorphins
ACh: function
voluntary motor movements
memory

* LOW levels ACh in Alzheimer's
Catecholamines: function
personality
mood
drive

* catecholamine hypot
* dopamine hypot



Remember: Cats have Moody Personalities because they don't Drive

Examples of catecholamines
norepinephrine
epinephrine
dopamine
Catecholamine hypothesis
Depression due to:

LOW levels norepinephrine
Dopamine hypothesis
Schizophrenia due to:

HIGH levels dopamine
Serotonin: function
hunger
temperature regulation
sexual activity
aggression
onset of sleep
Serotonin hypothesis
Schizophrenia & Autism associated with:

HIGH levels serotonin


Depression, PTSD, OCD associated with:

LOW levels serotonin
GABA: function
sleep
eating
seizure
anxiety disorder

* LOW levels GABA in Huntington's Disease
Glutamate: function
learning
long-term memory

HIGH glutamate in Huntington's & Alzheimer's
Medulla: function

[brainstem; hindbrain]
vital ANS functions:

circulation
respiration
Medulla: damage

[brainstem; hindbrain]
fatal
Pons: function

[brainstem; hindbrain]
transmits motor info from higher brain & spinal cord to cerebellum

integrates movements in R & L sides of body

respiration
feeding
sleep
Cerebellum: function

[brainstem; hindbrain]
balance
posture
coordinated & refined motor movements
Cerebellum: damage
[brainstem; hindbrain]
"ataxia"

lack of balance
severe tremors
drunken-like movements
Reticular Activating System (RAS): function

[midbrain]
screens incoming info & relays to higher centers of brain

arousal
Reticular Activating System (RAS): damage

[midbrain]
disrupts normal sleep-wakefulness cyle

possible permanent coma-like sleep
Thalamus: function

[diencephalon; forebrain]
"relay station" for all sensory input (EXCEPT olfaction)
Thalamus: damage

[diencephalon; forebrain]
Wernicke-Korsakoff syndrome
- caused by thiamine deficiney as a result of alcoholism
Wernicke-Korsakoff syndrome
caused by thiamine deficiney as a result of alcoholism

severe anterograde amnesia
retrograde amnesia
confabulation
Hypothalamus: function

[diencephalon; forebrain]
maintains body's internal HOMEOSTASIS

controls ANS & endocrine glands
mediates basic drives
regulates emotional expression
Amygdala: function

[limbic sys; telencephalon; forebrain]
controls emotional activities

mediates defensive-aggressive bheaviours

attaches emotions to memories
Amygdala: damage
[limbic sys; telencephalon; forebrain]
reduced aggressiveness

Kluver-Bucy Syndrome
- compulsive oral behaviours
- hypersexuality
- visual agnosia
Kluver-Bucy Syndrome
damage to amygdala

compulsive oral behaviours
hypersexuality
visual agnosia
Hippocampus: function

[limbic sys; telencephalon; forebrain]
consolidation of memory
Hippocampus: damage

[limbic sys; telencephalon; forebrain]
inability to form new memories
Frontal lobe: function

[cerebral cortex]
emotion & personality
planning
decision making
Cerebral cortex makes _____ % of the brain's total weight
80%
Frontal lobe: damage

[cerbral cortex]
changes in personality
loss of a sense of "self"
inability to carry out plans
Parietal lobe: function

[cerbral cortex]
somatosensory cortex

pain
pressure
heat
cold
Parietal lobe: damage

[cerbral cortex]
impairments in:

spatial orientation
touch
facial recognition

* can cause Gerstmann syndrome
Temporal: function

[cerbral cortex]
auditory perception
Occipital: function

[cerbral cortex]
visual perception
limbic system is primary associated with ___________
emotion
4 lobes of cerebral cortex
frontal
parietal
temporal
occipital
Basal Ganglia: function

[telencephalon; forebrain]
planning
organizing
voluntary movement
regulating motor actions
Basal Ganglia: associated disorders

[telencephalon; forebrain]
Parkinson's
Tourette's

Mania
Depression
OCD
Psychosis
hemispheric specialization
LEFT (dominant)
verbal activities
analytical, logical thought
positive emotional states

RIGHT
visual-spatial activities
e.g. facial recognition
spatial interpretation
memory for shapes
negative emotions
corpus callosum
major pathway for info travelling between hemispheres
2 branches of nervous system
central nervous system

peripheral nervous system
structures of CNS
brain
spinal cord
Autonomic Nervous System: function
controls internal glands/organs, including:

heart
bladder
stomach
endocrine glands
sympathetic nervous system: function

[autonomic nervous sys; peripheral nervous sys]
active during stress & excitement

promotes energy expenditure by:

inc blood sugar
accelerating breathing
raising blood pressure & heart rate
parasympathetic nervouse system: function

[autonomic nervous sys; peripheral nervous sys]
operates during states of relaxation

helps body conserve energy by:

slowing heart rate & blood flow
inhibiting glucose release
constricting blood vessels
suprachiasmatic nucleus (SCN): location & function
hypothalamus

mediates sleep-wake cycle
mediates circadian rhythms

*involved in Seasonal Affective Disorder
Broca's area: location & function

[premotor cortex; frontal lobe; cerebral cortex]
premotor cortex

speech production
Broca's area: damage

[premotor cortex; frontal lobe; cerebral cortex]
Broca's (expressive) aphasia

- difficulties in producing spoken & written language
Prefrontal cortex: function

[frontal lobe; cerebral cortex]
complex behaviours

emotion
semory
self-awareness
executive functions
Prefrontal cortex: damage

[frontal lobe; cerebral cortex]
pseudodepression

pseudopsychopathy

probs w/ abstract thinking, planning, decision-making
perseveration
apraxia
inability to perform skilled motor movements in absence of impaired motor functioning
anosognosia
inability to recognize one's own neurological sx or other disorder
Wernicke's area: location & function

[temporal lobe; cerebral cortex]
dominant temporal lobe

comprehension of language
visual agnosia
inability to recognize familiar objects
simultanagnosis
inability to see more than one thing or one aspect of an object at a time
prosopagnosia
inability to recognize familiar faces
contralateral representation
L side of brain controls R side of body, and vice versa
Hemispheric specialization
LEFT (dominant)
written & spoken language
logical & analytical thinking

RIGHT
spatial processing
creativity
facial recognition
"split-brain" patients
sever corpus callosum to control severe epilepsy

deficits in ability to verbally identify info presented to RIGHT hemisphere only.
types of colour blindness
Trichromats
normal colour vision

Dichromats
lack 1 of 3 pigments
usually red-green blind

Monochromats
no colour vision
3 theories of emotion
James-Lange theory

Cannon-Bard theory

two-factor (cognitive) theory
James-Lange theory of emotion
emotions represent perceptions of bodily reactions

e.g. you are afraid because your knees are skaking

support from quadriplegics experiencing less intense emotions following injury
Cannon-Bard theory of emotion
emotional & bodily reactions to stimuli occur simultaneously
Two-factor (cognitive) theory of emotion
emotion a consequence of:
1. physiological arousal
2. cognitive interpretation
3. enviornmental context

support from Schachter & Singer's epinephrine study
general adaptation syndrome (Selye)
human response to stress mediated by adrenal-pituitary secretions & involves 3 stages

alarm reaction
resistance (ACTH/cortisol)
exhaustion
gonadotropic hormones
estrogen (ovaries)
progesterone (ovaries)
testosterone (testes)
androstenedion (testes)
androgens
testosterone
androstenedione
closed-head injury
loss of consciousness (coma)
post-traumatic (anterograde) amnesia
sometimes retrograde amnesia
cognitive probs if PTA > 24 hrs
hyperthyroidism vs. hypothyroidism
HYPER (Grave's Disease)
fast metabolism
high body temp
inc appetite w/ weight loss
nervousness

HYPO
slow metabolism
dec appetite w/ weight gain
lethargy
depression
apathy
Broca's Aphasia
"expressive, motor, and nonfluent aphasia"

slow speech w/ difficulty
poor articulation
omissions
anomia
probs repeating phrases

comprehension of language OK
aware of deficits
frustration & depression
Wernicke's Aphasia
"receptive, sensory, and fluent aphasia"

probs understanding
speech devoid of content
anomia
paraphasia

unaware that speech is meaningless
Conduction (Associative) Aphasia
damage to structure that connects Wernicke's & Broca's areas

anomia
inability to repeat words

know what they want to say, but have difficulty
Global Aphasia
total or near-total loss of language

some automatic speech
Transcortical Aphasia
lesion that isolates Broca's leads to:
transcortical MOTOR aphasia

lesion that isolates Wernicke's leads to:
transcortical SENSORY aphasia