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233 Cards in this Set

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Roles and responsibilities of the athletic trainer?

1.) injury prevention


2.) injury evaluation


3.) immediate and emergency care


4.) organizational qualities

1.) injury prevention?

- provide safe environment


- provide care when injury occurs


- ensure that athlete conditioned to participate


- equipment
- maintenance of protective equipment

2.) injury evaluation?

- recognize nature and extent of injury


- understand pathology of sport related injuries


- injury management


- know when to refer

3.) immediate and emergency care?

- administration if appropriate first aid


- avoid delaying injury management


- be aware of coaching/peer pressure


- do not allow minor injuries to become major


- activation of EAP

4.) organizational qualities?

- education coaches and athletes


- developing confident medical team - consistency


- cooperate with coaching staff


- establish rapport with team


- SOAP - subjective (patient history), Objective (observation), Assessment (examination), Plan (rehab)

Personal and professional qualities of trainer?

- timeliness


- professional appearance


- effective communication


- lack of bias and ethical practice


- polite to referees


- sense of fair play


- gossiping and relationships



what are the expectations of you?

- recognize limitations


- prevent unnecessary delays


- link btw athletic program and medical community for implementation of injury



What are your limitations?

DO NOT:


- diagnose


- share info without consent


- prescribe meds


- reduce fractures


- move anybody who can not move

What are modifiable and non-modifiable intrinsic risk factors? example?

Mental and psychological aspects that can result in injury or harm towards individual




eg modifiable - skill level, motivation, discipline, previous experience in sports, risk taking behaviours


eg non-modifiable - age, sex, previous injuries, innate intelligence


other eg. strength, endurance, joint, structural ****, reaction time, timing, speed, agility

Extrinsic risk factors?

aspects of a competition from which a person exposes themselves to harm or injury




modifiable eg. type of equipment, amount of time played, environment (crowd control, footwear)


non-modifiable eg. time of season, weather conditions, time of day

Why do research?

collection of large amounts of data helps to identify trends


can help reduce injuries


- data is used to modify rules


- assisting coaches and players to understand risks

Definition of emergency?

an unforeseen combination of circumstances or the resulting state that calls for immediate action


- sudden bodily alteration likely requiring immediate medical attention


- eg. ruptured appendix


- usually distressing event or condition, can often be anticipated

How do you prepare for an emergency?

- set up EAP b4 EMG


- review it


- personnel and roles


- location and contact info


- call sheet

How do you determine an emergent situation?
determined by mechanism of injury, player movements on field and observation upon approach
What must be done immediately upon on field injury?
On field assessment - first step for proper injury management
What consists of an on field assessment?

1. Primary survey


- determine presence of life threatening injuries or conditions


2. secondary survey


- determines presence of other issues that are not life threatening NOW, but may become so

Some life threatening conditions?

- Airway obstruction


- no breathing


- no circulation


- profuse/deadly bleeding


- shock

Some conditions that require immediate action?

- Hyperthermia


- Hypothermia


- Head injury


- Fracture or dislocation of spine or long bone
- Serious eye injuries


- Athlete unwilling to move or cannot support own body weight

What is shock?

Heart not able to exert enough pressure to circulate oxygenated blood to vital organs due to:


- damaged heart


- low blood volume


- blood vessels dilation which causes pooling away from vital organs that takes oxygen away from cells

Types of shock?

hypovolemic - dec. blood vol and BP


respiratory- lungs cannot supply enough O2


neurogenic- dilate of peripheral vessels due to CNS truma


metabolic- complication of untreated diabete


psychogenic- temp dilation of blood vessels & decreased blood to brain


cardiogenic- heart incapable of cycling blood


septic- severe infection


anaphylactic*- dilation of peripheral blood vessels due to severe allergic reaction. hypotensive but tachycardic

signs and symptoms of shock?

- rapid, weak pulse


- shallow, rapid breathing


- pale, cool, clammy skin


- drowsy, sluggish


- dizzy


- dilated pupils


- thirsty

How manage shock?

- activate EMS


- obtain and maintain open airway


- manage complications or conditions


- maintain body temp


- elevate legs in MOST situations


- reassure and keep calm


- monitor and record vital signs

When do you activate an EAP?

when its beyond your scope of knowledge


- unconscious


- no circulation


- spinal, eye injury


- internal trauma


- head trauma


- deadly bleeding

What type of decisions do u need to make when doing an on field assessment?

Injury severity? is it life threatening?


What type of first aid required?


Is a medical referral necessary?
Does this athlete need to be transported?

What is primary survey protocol?

- safety first - safe enviro? stabilize head


- check- unresponsiveness/airway, mouthguard?


- call- activate EAP, provide dispatcher with maximal info possible


- care- cpr? bleeding?

Major bleeding?

Hemorrhage - excessive discharge of blood internally or externally


- venous- dark red; continuous flow


- capillary- bubbles up; oozes redish flow


- arterial- bright red spurting flow

How do you manage bleeding?

RED


- rest


- elevation


- direct pressure


and treat for shock

Secondary survey?

- vital signs


- conduct SAMPLE


- head to toe examination


- conscious vs unconscious


perform basic neurological exam


- MSC - motor sensory circulation

SAMPLE?

- Sign and symptoms


- Allergies


- Medications


- Past medical history


- Last meal


- Events prior

How do you check level of consciousness?

AVPU


- awake?


- does the patient respond to verbal stimulus?


- does the patient respond to painful stimuli?


- is the patient completely unresponsive?

when doing a secondary survey what do you do if the patient is unconscious?

Do vitals

When doing a secondary survey what do you do if the patient is conscious?

Do SAMPLE followed by a set of vitals

What are vital signs of a secondary survey?

- AVPU


- Pulse check


- respiration rate and quality


- unresponsive- check by look,listen, feel


- responsive- check unobtrusively


- skin colouration/temp


- pupil responsiveness


- if pupils dont accomadate to light, probs brain damage


- blood pressure but not really

What are you scanning for in an head to toe body scan?

depressions, contusions, abrasions, penetrations, bruising, lacerations, swelling, tenderness, instability, crepitus, blood

MSC?

motor sensory circulation test


- grip strength, wiggle toes


- numbness and tingling


- pinch nail beds

What is risk?

Potential of something happening that will have an impact



How to manage risk?

- Goal is not only to prevent harm, but to educate and promote good


- Priority 1 is to ensure participant safety


- Consult those around you


- Should be a group effort amongest players, staff, coaches, refs, medical team

2 types of legal action?

1. Criminal suit


- robberies, assaults, murder


2. Civil suit


- non-premeditated offences


- most common litigation in sport

Liability?

Being legally responsible for the harm one causes another person



Tort?

A legal wrong


May emanate from:


-Nonfeasance- fail to perform legal duty(fail to refer)


-Malfeasance- performs action that is not his/hers to legally perform


-Misfeasance- performs an action incorrectly that the person has legal right to do

Commission?

an act unlawful and intended to cause harm

Omission?

failure to perform a specific legal duty

Negligence?

Not providing reasonable or ordinary care when people in the same situation as u would

Foreseeability?

condition where danger is apparent or should have been apparent which ultimately resulted in unreasonable, unsafe conditions

What does shared responsibility mean?

The athlete has a responsibility to report injuries that occur and provide relevant medical history

What is needed for a negligence suit to be successful?

- prove that there was a duty to exercise reasonable care


- prove that there was duty that was breached


- establish a connection to link the breach of duty and the injury

What are some potential areas of negligence?

- Supervision


- Instruction


- Unsafe facilities


- Defective equipment


- Transportation

Ways to avoid negligence?

- Fulfilling duty of care and contractual obligations


- Establish and follow an EAP and initiate emergency process if required


- Communicate


- Be familiar with athletes' medical histories


- Know your limitations


- Maintain confidentiality

Do you need consent from someone before doing first aid?

yes, ensure athlete wants treatment and explain what the treatment is


- Legal consent may only be granted by persons 19 years and older, otherwise parental consent required

Informed consent?

You explain ALL the risks and benefits of the treatment/assessment before u begin

Explicit consent?

Athlete comes to you for assistance or they agree to first aid

Implied consent?

Individual is unable to give explicit consent but required it bc of an emergency


- unconscious/unresponsive

Good samaritan act?

Protects person providing emergency care to individual from being liable for negligence if something goes wrong


- doesnt apply to people employed or paid to provide care

Skull fracture?

Etiology: direct blow or indirect force that travels from mandible to skull




usually creates epidural, subdural hematoma or intra-cranial hemorrhaging

Signs and symptoms of skull fracture?

- severe headache and nausea


- palpation may reveal defect or deformity


- may be blood in middle ear, ear canal, nose, around eyes or behind ear (battle sign)


- cerebrospinal fluid may also appear in ear and nose


- inability to see or smell




IMMEDIATE HOSPITALIZATION

Cerebral contusion ?

Etiology: focal injury to brain that involves small hemorrahages or intracranial bleeding in cortex, generally when head strikes stationary object




Signs: depends on extent


- dizziness, nausea, LOC followed by talkative state




management: hospital, physician clearance

Epidural Hematoma?

Etiology: direct blow to head for fracture, damages superficial tissue and meningeal artery


- blood accumulation and hematoma formation occurs rapidly due to arterial pressure




Signs: LOC followed by lucidity, showing few sign


- gradual progression of: head pains, dizziness, nausea, dilation of one pupil, deterioration of consciousness, neck rigidity




Management: requires urgent neurosurgical care; relieve pressure to avoid disability

Subdural Hematoma?

Etiology: result of acceleration/deceleration forces that tear vessels that bridge dura mater and brain


- can be acute (rapidly progressing) or Chronic(due to venous bleeding)




Signs: LOC does not occur


- sometimes LOC and dilation of 1 pupil


- headaches dizziness




Management: immediate medical attention

Malignant brain edema syndrome?

Etiology: occurs in young within mins to hours following head injury


- intracranial clot resulting in diffuse brain swelling


- swelling result of hyperemia or vascular engorgement - results in increased pressure




signs: rapid neurologic deterioration, then coma




management: immediate hospitalization

Decorticate posturing?

- indicative of damage to neural pathway btw brain and spinal cord


- posturing may be uni or bilaterall


- FLEXED POSTURING, START FLEXING BODY TOGETHER


- stiff body with bent arms and clenched fists


- legs internally rotated

Decerebrate posture?

- typically associated with severe brain trauma


- severe muscle spasming of neck and back


- posture may be uni or bilateral, just in arms


- extension of body


- FOREARMS AND ARMS PRONATED FISTS


- feet planterflexed


MORE SEVERE

Most frequent facial fracture?

nasal

Mandibular dislocation?

Etiology: TMJ joint


- MOI is generally a blow to an open mouth from the side




signs: dislocated jaw present in locked or open position w/ROM minimal along w/poor occlusion(sealing)




Management: cold application, elastic wrap immobilization and reduction


- physician clearance needed before return

Mandibular fracture?

Etiology: direct blow




signs: deformity, loss of occlusion, pain with biting, bleeding around teeth




management: immobilization with elastic wrap and ice


- refer to hospital for reduction and fixation

Maxillary and zygomatic fractures?

MOI- direct blow




signs: deformity, nosebleed, double vision, numbness




M: controls swelling and maintain airway


- manage open wounds


- assume potential concussion

Orbital fracture?

E: blow to cheek or eyeball forcing it posteriorly




S: diplopia, restricted eye movement, downward displacement of eye, swelling




M: ice and advise not to blow nose

Nasal fracture?

E: direct blow or from side




S: profuse bleeding, pain, crepitus(crackling of bones), swelling




M: control bleeding


- rule out concussion

Dental fracture?

E: impact to jaw or direct trauma




S: uncomplicated fractures produce fragments without bleeding


- complicated fractures produce blood with lots of pain




M: bleeding can be controlled with gauze


- no biggie just chill nigga can get fixed later


- mandibular fractures and concussion must be ruled out

Lacerations?

E: result of a direct impact and indirect compressive force or contact with sharp object




S: profuse bleeding, localized swelling, localized pain




M: REDs to control bleeding


- clean wound and prevent contamination


- look for skull fracture


- ster-istrips

Tooth subluxation, Luxation or avulsion?

E: direct blow




S: tooth may be loose


Sublux- loose within socket, lil/no pain


Luxation- tooth displacement w/o fracture


avulsion- knocked out




M: sub- referral with 48 hours


sublux- immediate follow up


avul- milk or saline within 2 hours, if tooth driven inwards refer to dentist immediately

Eye injury assessment and observation?

assessment:


- transport in recumbent position


- both eyes should be covered, movement in unaffected eye will cause movement in the other eye




observation: swelling discolouration, hemorrhaging, deformity

Orbital Hematoma?

E: blow to area surrounding eye




S: swelling and discoloration




M: ice for 30 mins


- dont blow nose after acute eye injury


- monitor concussion

Hyphema?

E: blunt blow to eye, can lead to serious problems with lens, retina




S: blood collect in anterior eye, blood may turn pea green


- vision partially or completely blocked


- drowsiness




M: physician immediately

Ruptured tympanic membrane (eye injury)?

E: fall or slap to unprotected ear or sudden underwater pressure variation




S: complaint of loud pop, followed by pain in ear, nausea, vomitting




M:physician, infection can occur so gotta monitor

Auricular hematoma? (cauliflower ear)

E: compression or shear injury to ear




S: tearing of overlying tissue away from cartilage


- Hemorrhaging and fluid accumulation




M: proper ear protection to prevent


- ice



How to prevent spinal injuries?

Cervical - strengthening of cervical or para spinal musculature


- maintain functional ROM




Thoracic/lumbar - avoid repetitive stresses


- correct biomechanical abnormalities


- correct technique




spine injuries occur when forced beyond normal ROM

Muscular strains?

E: usually with hyperflexion/rotational force or hyperextention/rotational force


- chronic strain associated with posture




S: pain may be diffuse or localized


- pain with active motion and/or stretching, spasms, disability




M: RICE, activity modification, gentle stretching/ROM

Ligamentous sprains?

E: generally same as strain, more violent (eg. whiplash)




S: same as muscle strain but lasts longer


- Localized pain and tenderness over transverse and spinous processes


- decreased passive and active ROM




M: RICE, NSAIDs

Cervical fracture?

E: generally axial load w/ some degree of cervical flexion




S: neck point tenderness, restricted motion, cervical muscle spasm, cervical pain




M: stabilize, spine board

Lumbar fracture?

E: compression fracture or of spinous or transverse processes


- compression fractures are usually the result of trunk hyperflexion or falling from a height



S: X-rays


- point tenderness


- palpable defects


localized swelling and guarding




M: X-ray


- transport with extreme caution and care to minimize movement

Brachial plexus neuropraxia?

E: stretching or compression of brachial plexus
- disrupts peripheral nerve function w/o degenerative changes




S: burning sensation, numbness, tingling and pain from shoulder to hand


- some loss of function in arm and hand




M: strengthening and stretching program, return to activity ones signs are normal, padding to limit neck ROM

Spondylolysis and Spondylolisthesis?

E: lysis refers to degeneratio of vertebrae due to congenital weakness (stress fracture)




listhesis- slipping of one vertebrae above or below another, often associated with each other




S: lysis begins unilaterally


- pain and persistent aching, low back stiffness with increased pain after activity, frequent change in position, localized tenderness




M: strengthening and stabilization exercises


- increased susceptibility to lumbar sprains and strains

Define a concussion?

- Results from complex pathophysiological process process affecting brain


- subset of a traumatic brain injuries


- functional injuries- perturbations of cellular or physiological function - iconic shifts, metabolic changes, impaired neurotransmission

Mechanism of injury for concussion?

Cause by biomechanical forces to body


- direct or indirect blow, to head, neck or face


- stumbly, clumsy, unsure of game situation, can recall event before or after contact

What does the acute and persistent phase consist of?

Acute: 0-10 days post injury


- increased calcium levels


- decreased oxidative metabolism


- cells cannot repair until Ca normalizes




Persistent:


- axonal damage


- cell death if Ca level remain elevated


- glucose metabolism can be decreased for up to month

Define neuroautonomic disruption

- uncoupling of autonomic NS and cardiovas system


- uncoupling can be monitored with HRV


- dec. in HRV due to increase to SNS


- dec. in cerebral blood flow

What are the 3 categories of Signs and symptoms?

somatic




cognitive




neurobehavioural

Somatic signs and symptoms of concussions?

- headache


- nausea


- sensitive to light and/or noise


- numbing or tingling


- balance/coordination probs




Somatic- relating to the body

Cognitive S&S of concussions?

- Feeling slowed down


- feeling in a fog


- difficulty concentrating


- difficulty remembering




Mental aspects

Neurobehavioural S&S of concussions?

- Sleeping more or trouble sleeping


- Drowsiness


- Fatigue


- Sadness/depression


- nervousness


- irritable

What is the Glasgow coma scale?


And the 3 categories?

Used to determine conscious state of person




3 categories:


- eyes response


- verbal response


- motor response




the higher the score the better off the person is

What is a secondary impact syndrome?

Occurs when athlete suffers a concussion before the first one is fully resolved




Can be catastrophic:


- Increase symptom severity


- increase duration of symptoms


- permanent brain damage


- death

What to do when concussions occurs?

- Remove player from play ASAP


- SCAT/KD


- wait 15 mins to let the player calm down


- let coaching staff know


- refer for medical follow up


- document injury

What concussions symptoms are an emergency?

- Seizures


- Unusual behaviour


- repeated vomitting


- Slurred speech


- Increase confusion


- Cant recognize people or places

After concussion occurs what information should you provide to the injured athlete in terms of recovery?

- Limit mental and physical activity as this can prolong recovery


- Limit screen time (texting, tv, computer, video games)


- avoid loud stimulating places


- no alcohol


- get cleared by doctor before RTP

When can an athlete RTP?

- Gradual RTP protocol


- starts once athlete is asymptomatic


- 6 steps, if an S&S arises, rest for 24 hours and retry the step

How to help prevent a concussion?

- ensure proper fitted equipment


- equipment in good condition


- mouth guards


- fitness level


- proper technique

Where does the 1st rib lay? and from where is it best palpated?

Below the clavicle and from a superior approach

Which rib protects the spleen?


Which rib protects the Kidney?

10


12

What is happening to the ribs when we inhale?

Ribs are pulled up and forward; increase in overall A/P diameter


- ribs 1-6 increase in anteroposterior dimension


- ribs 7-10 we mainly see an increase in lateral dimension

How many quadrants are the abs divided into?

4


right and left upper quadrant


right and left lower quadrant



How many regions are the abs divided into?

9


right/left hypochondriac, epigastric regions


right/left lumbar, umbilical region


right/left iliac, hypogastric region

What consists of a general assessment of abdominal injuries?

Check for discoloration, swelling, and deformities


- protusions, bloat, swelling?


- are abs tight and guarded?


monitor vitals

S&S of thoracic pain?

- Cyanosis (bluish color of lips, skin, fingers)


- dyspnea


- chest pain with breathing


- distended neck veins


- reduced chest movement


- shifting of trachea with breath


- couching blood


- shock



S&S for abs and pelvis?

- severe ab or pelvic pain


- point tenderness


- spasm of ab muscles


- blood in urine or stool


- nausea


- sensation of weakness


palpable defect or deformity

Rib fractures?

E: direct blow


- prolonged repetitive movements


- violent muscular contraction (coughing/sneeze)




S: pain with INSPIRATION


- point tenderness


- deformity with palpation


- coughing up blood




M: refer for x rays


- monitor for complications


- support and rest; brace

Whats a flail chest?

When 3 consecutive ribs r injured

Pneumothorax?

Pleural cavity becomes filled with air, negatively pressurizing cavity, causing lung to collapse

Tension Pneumothorax?

Pleural sac on one side fills with air displacing lung and heart, compressing opposite lung

Hemothorax?

Blood in pleural cavity causes tearing or puncturing of lungs or pleural tissue

Traumatic asphyxia?

result of violent blow or compression of rib cage

Common S&S of lung injuries?


Management?

- Difficulty breathing (dyspnea)


- breathing cessation (Apnea)


- Cyanosis, pain, hemoptysis and shock




M: medical emergencies


- treat for shock


- monitor vitals, perform rescue breathing or CPR



Hyperventilation?

E: rapid rate of overbreathing due to anxiety or asthma


- less CO2 compared to O2




S: rapid breathing that induces panic


- belching, bloating, confusion, light headed




M: calm person down


- breath through 1 nostril or pursed lips

Hypertrophic cardiomyopathy?

Thickening of cardiac muscle w/no increase in chamber size

Marfan's syndrome?

Abnormality in connective tissue results in weakening of aorta and cardiac vessels

Sudden cardiac death syndrome?

E: Coronary artery and peripheral artery disease


- right ventricular dysplasia(enlargement); cardiac conduction abnormalities, aortic stenosis (narrowing of aorta)


- drugs, alcohol, intracranial bleeding, obstructed respiratory disease




S: usually no signs prior to death


- chest pain, heart palpitations, syncope, nausea, profuse sweating, SOB, fever




M: Counseling and screening are critical in early identification and prevention


- history of heart murmurs, chest pain during activity

Commotio Cordis?

E: cardiac arrest due to impact to chest


- young athletes at risk bc chest pliability




S: ventricular fibrillation




M: resuscitation is rarely successful


- AED

Kidney contusion?

E: result of external force




S: shock, nausea, vomitting, muscle guarding




M: advice to check for hematuria(blood in urine)

Ruptured spleen?

E: direct blow


- mononucleosis




S: shock, ab rigidity and spasm, pain, nausea


- Kehr's sign




M: Medical emergency


- treat for shock

Contusion of abs?(solar plexus)

E: direct blow to ab or indirectly from falling


- diaphragm going into temporary spasm




S: localized tenderness, swelling, difficulty breathing




M: calm individual


- rule out internal injuries


- short breaths in and long out


- loosen tight clothes


- DO NOT PUMP LEGS INTO ABS

Ab pain- "side stitch"?

E: most common cause of ab pain with exercise


- worst after eating




S: described as ache or stitch on either costal angle




M: stop activity and stretch arm overhead


- forced expiration again pursed lips

Hernias?

E: Protusion of ab viscera through portion of ab wall


- heavy lifting or ab blow




S: prolonged pain and discomfort, deformity that appears w/ coughing


- weakness or pulling sensation in groin




M: refer to physician for surgical consult

Appendicitis?

E: chronic or acute inflammation of appendix


- starts as gastric complaint, then develops to red swollen vessel




S: mild to severe pain in lower ab, w/ nausea, vomitting and low grade fever


- pain in lower right ab




M: surgery

Scrotal contusions? getting sacked lol it hurts so bad

E: result of blunt trauma




S: hemorrhaging, fluid effusion, muscle spasm, severe pain, nausea




M: flex knees up and breathe in short breathes


- check if both nuts are there LOOOL

Ruptured bladder?

E: force to lower ab


- hematuria often associated with contusion of bladder during running




S: pain, discomfort of low ab, ab rigidity, nausea


- inability to urinate will present in case of ruptured bladder




M: physician


- encourage to urinate frequently

Radiation?

Heat loss from warmer object to cooler object - indirectly

Conduction?

Warmer body gives off heat to cooler object, directly

Convection?

Cooler air blowing over body will cool off the body

Evaporation?

perspiration from on surface of skin and evaporates, carrying the heat with it

What is the primary way to dissipate heat in temperatures greater then 20 degrees c?

Evaporation

Primary way to dissipate heat in temps less then 20 degrees?

conduction and evaporation

Who is at risk for heat illness and hyperthermia?

- Very young and old


- obese or large muscle mass


- poor conditioning and acclimatization


- previous history of heat illness


- sleep deprived


- dehydration


- acute illness (fever)


- chronic illnesses (alcoholic, cardiac disease)


- wheelchair athletes

Heat syncope? E,S,M?

E: individual abruptly stops exercising and blood pool - blood not being returned to brain or heart




S: lightheadedness or fainting




M: elevate legs, cool down with cold towels, rehydrate and remove from heat

Muscle cramp?

E: lack of hydration or imbalance btw water and electrolytes


- results in involuntary muscle spasm


- painful involuntary skeletal muscle contraction


S: painful/violent muscle ramp in calf or abs




M: rehydrate, ice cramp, passive stretch

Heat exhaustion?

E: bodys ability to dissipate heat compromised and core temp increases




S: extreme weakness, sweating, light headed, tachycardia, cramping




M: rehydrate frequently, cool towels, sponges, ice packs on groin, weak pulse

Heat stroke?

E: body's inability to dissipate heat and results in core temp > 40




S: sudden collapse, LOC, flushed, hot skin, shallow breathing




M: 911, lower core temp, cold towels, remove from heat, DO NOT IMMERSE IN WATER, strong pulse

What is exertional hyponatremia?

Disorder involving fluid-electrolyte imbalance


- results in low sodium concentration


- disrupts osmotic balance across the blood brain barrier and causes rapid influx of water into brain


- brain swelling and weird neurological responses

S&S of exertional hyponatremia?

- confusion, seizure or coma - can eventually lead to death bc braintem ruptures


- bloating or nausea


- headache, nausea, vomitting, wheezy breathing, swollen hands and feet, fatigue


- respiratory arrest, coma, permanent brain damage and death

What factors have an affect on hypothermia?

- Low temp factors


- chill factor


- dampness factor




temp in conjunction with wind and dampness can increase chances




heat lost exceeds heat production

Hypothermia? ESM?

E: bodys ability to produce heat impaired and core temp falls less the 35 degrees




S: shivering, numbness, lack of coordination, confused or unusual behaviour, slurred speech




M: may be medical emergency, remove from cold, remove wet clothing, re-war slowly, give warm liquids and monitor vitals

Degrees of hypothermia?

- intense, uncontrollable shivering


- violent shivering, difficulty speaking if conscious


- less shivering, muscular rigidity, decreased coordination, confusion to amnesia


- unconscious and unresponsive, pulse erratic


- heart and lungs fail, hemorrhaging and death

Frost bite or nip?

E: fluid btw cells crystalizes and expands causing damage to tissues and vessels




S: swelling, pain and tingling, numbness later, skin appears waxy, hard, discoloured (blue) or feels cold




M: remove from cold, rewarm SLOWLY, DO NOT rub area

Altitude sickness?

- As height increases, O2 uptake decreases and results in decrease in performance


- body compensates through tachycardia and hyperventilation



Acute mountain sickness?

- Anorexia


- nausea


- vomitting


- insomnia


- dizziness

High altitude cerebral edema?

- headache


- disorientation


- loss of coordination


- memory loss

High altitude pulmonary edema?

- Chest tightness


- persistent cough


- frothy sputurm


- suffocation

Where is the best place to shelter during lightening storm?

Indoors




and avoid large trees, flag/light poles, standing water, pools, telephones, showers and metal objects


- last resort find a car, ditch, valley

Diabetes ESM? types of diabetes?

Type 1- insulin dependent


- typically occurs in individuals under age 35


type 2- non insulin dependent


- occurs in all ages, becoming more prevalent in younger kids due to obesity




E: decrease in insulin secretion




M: monitor and control glucose levels


- insulin


- vigorous exercise increases peripheral insulin action and enhances glucose tolerance

Insulin shock?

E: to much insulin, not enough blood sugar




S: tingling in mouth, hands, physical weakness, headaches, ab pain


- normal or shallow respiration, rapid heart rate




M: adhere to careful planned diet, snacks before exercise

Diabetic coma?

E: loss of sodium, potassium and ketone bodies through excessive urination (ketoacidosis)




S: labored breathing, fruity smelling breath, nausea, thirst, dry mucous membranes, confusion




M: Early detection is critical as this is life threatening


- insulin injection may help

Epilepsy?

Paroxysmal disorder (physical dysfunction), periods of altered consciousness, motor activity




E: sometimes genetics


- brain injury or altered brain metabolism




S: periods of altered consciousness, motor activity, sensory phenomena or inappropriate behavior




M: physician clearance prior to participation


- must be careful with activities involving changes in pressure

Exercise induced asthma?

E: bronchocontriction


- aggravated in cold, dry, polluted or allergenic filled air




S: coughing, wheezing, excessive spitting, dyspnea, chest tightness




M: avoid triggers and warm appropriately


- cover nose and mouth to warm and humidify air

Runners' anemia (hemolysis)?

E: caused by impact of foot as strikes the surface


- impact destroys normal erythrocytes within vascular system




S: mildly enlarge cells, increase in immature RBCs and negatively affect hemoglobin




M: reduce distance or mileage

Female athlete triad?

E: relationship btw disordered eating, amenorrhea and osteoporosis


- seen in females driven to meet standards of sport or to meet a specific athletic image to attain goals




S: disordered eating- bulimia and anorexia


- osteoporosis- premature bone loss in young women, inadequate bone development




M: prevention is key; identify and educate

What are the pain qualities for nerve pain, bone, fracture, vascular?

nerve- bright, burning, specific distribution




Bone- deep, boring, localized, nagging




fracture- sharp, severe, intolerable




vascular- diffuse, throbbing, generalized, may be referred

Pain qualities for muscle, neuropathic, ligament, somatic?

Muscle- dull, aching, generalized, aggravated




neuropathic- injury or pathology PNS or CNS




ligament- dull/ aching




somatic- chronic, aching pain that is inconsistent or inexplicable

What are some possible red flags?

- Severe unremitting pain


- pain unaffected by meds or position


- severe night pain


- severe pain with no history or trauma


- severe spasm


- bowel/bladder changes


- changes in vision


- swallowing or speech changes


- falling down


- SOB, heavy chest

Ideal posture?

- Slightly anterior to lateral malleolus


- slightly ant to fibular head


- at greater trochanter


- at acromion


- at external auditory meatus (EAM)

What does active-examining test?

- Willingness to move


- Tests function of both contractile and inert tissues through available ranges


- motion control


- patterns of restriction


- muscular power and adopted adaptations


- ask for permission


- clear joints above and below


- examine unaffected side first and painful movements last

What does passive examining test?

Assessment done by trainer


- patient must be relaxed


Goal is to note any limitation or presence of pain


Passive ROM examines inert structures


- bones, ligaments, fascia, bursae, nerves, nerve roots

Passive-normal end feels?

bone to bone


soft tissue approximation


firm/capsular (tissue stretch)


- abrupt, hard, firm endpoint



abnormal end feels?

- Springy block (rebound)


- boggy (swelling)


- Empty (non or arrested)


- Spasm (guarding)


- loose (extreme hypermobility)

Benefit of resisted examination?

Manual muscle testing is one to determine if contractile structure have been affected

- also exams innervation


Contractile tissue - what are the 4 classic patterns?

1.) movement is strong and pain free


- muscle and nervous tissue intact and not cause of discomfort


2.) Movement is strong and painful


- Local lesion in muscle or tendion (1/2nd degree strain)


3.) Movement is weak and painful


- severe lesion around joint


- caused by reflex inhibition secondary to pain


4.) movement is weak and pain free


- 3 degree strian or neural involvement

Sources of muscular weakness?

- strain


- pain inhibition


- peripheral nerve injury


- nerve root lesion (myotome)


- upper motor neuron lesion


- tendon pathology


- avulsion


- psychological affect

Why palpate?

can differentiate btw tissue quality


- determine joint tenderness


- determine variations in temp


- note any alterations in sensation or presence of crepitus




begin away from injury and move towards

Define trauma?

Physical injury or wound produced by internal or external force

What is mechanical injury a result of?

Force or mechanical energy that changes the state of rest or uniform motion of matter

Common MOIs?

- all out exertion


- contact


- striking or throwing projectiles


- propulsion of the body through the air


- repetition of movement



Load?

external force acting on body causing internal reactions within tissues

Stiffness?

Ability of tissue to resist load

Stress?

internal resistance to load

Strain?

Internal change in tissue (length) resulting in deformation

Direct impact?

Injury occurs at the point of impact

Indirect impact?

injury occurs at point away from contact

what are the 5 types of tissue loading?

Compression




Tension




Shearing




Bending




Torsion

Describe compressive tissue loading

force that results in tissue crush - aka compression


- 2 forces applied towards each other




eg. standing, burst fracture of spine

Describe tension tissue loading

Forces that pulls and stretches tissue




eg. ligaments or muscles, hamstring strain

Shearing

Force that moves across the parallel organization of tissue


Stress causing 2 opposing bones that displace one each other




eg. Spondylolisthesis

Bending tissue loading?

4 points- 2 forces act at opposite ends of structure



3 points - 3 forces, 2 on ends and 1 in middle




eg. bone

Torsion?

twisting in opposite direction from opposite ends




twisting force that causes tissue to fail




eg. ACL

When does an injury become chronic?

When it doesnt heal properly

Impingement?

Pinching of intervening tissue btw 2 bony structures




eg. subdeltiod bursitis

Friction?

Reptitive frictino btw 2 structures or an intervening structure (bursa)




eg. illiotibial band friction syndrome

injuries are classified according to what?

- Stage of injury or healing




- Severity of the injury




- Type of tissue damaged/injured




- type of mechanism

1st degree muscle strain?

Some fibers stretched or torn, Full ROM but painful

2nd degree muscle strain?

multiple fibers torn


- active contraction painful


- divot is palpable, some swelling and discolouration

3rd degree strain?

Complete rupture of muscle or musculotendinous junction


- significant impairment


- great deal of pain

Abnormal muscle contraction is the result of...?

Failure in reciprocal coordination of agonist and antagonist




Electrolyte imbalance due to sweating or strength imbalance

Muscle gaurding?

muscles within effected area contract to immobilize area to minimize pain


- involuntary muscle contraction

What is a clonic and tonic muscle spasm?

Clonic- alternating involuntary muscular contractions and relaxations in quick succession




Tonic- rigid contraction that lasts a period of time

What is a contusion?

Bruise that results from a sudden traumatic blow that compresses the soft or boney tissues


- creates hemorrhaging


- chronically inflamed and contused tissue may result in generation of calcium deposits (myositis ossificans)

Tendinitis?

tenderness due to repeated microtrauma and degenerative changes


- inflammation of tendon


RESTTTT

Tendinosis/tendonopathies?

due to improper healing of tendinitis


- visibly swollen with stiffness and restricted motion

Tenosynovitis?

inflammation of synovial sheath




Acute- rapid onset, crepitus and diffuse swelling


Chronic - thickening of tendon with pain and crepitus




often occurs in flexor tendon of digit and biceps tendon

How does a ligamentous sprain occur?

Traumatic joint twist that causes stretching or tearing of connective tissue

Grade 1 sprain?

Some pain, minimal loss of function, no abnormal motion, mild point tenderness

Grade 2 sprain?

Pain, moderate loss of function, swelling and instability with tearing and separation of ligament fibers

Grade 3 sprain?

Extremely painful


- loss of function


- severe instability and swelling

Subluxation?

Breif, transient injury involving partial dislocation and spontaneous joint relocation

Dislocation?

disarticulation of joint


- stablizing structures of joint are disrupted


- common in fingers and shoulder



Separation?

separation of fibrous joint due to stretching or tearing of supporting tissues

Bone fracture S&S?

S: deformity, pain, point tenderness, swelling, pain on active and passive movements

stress fractures?

overload due to muscle contraction, altered stress distribution due to muscle fatigue, changes in surface

3 types of bone fractures?

linear - line parallel to bone


transverse- line perpendicular to bone


spiral


oblique

3 major stages of healing?

1. inflammatory phase: 1-4 days


2. fibro-plastic repair phase : 3 days- 6 weeks


3. maturation- remodeling phase: upto 3 years

Goal of leukocytes and phagocytic cells?

- protect


- localize


- decrease injurious agents


- prepare for healing and repair

inflammatory phase is characterized by SHARP. What does sharp stand for

- swelling


- heat


- altered function


- redness


- pain

Process of clot formation?

injury to cell -> chemical mediators liberated -> vascular reaction -> platelets and leukocytes adhere to vascular wall -> phagocytosis



What does histamine do?

First to arrive at wound


- causes vasodilation and changes cell permeability owing to swelling

Chemical mediators?

- impact adherence along cell wall


- increase permeability locally for fluid and protein passage


- facilitates exudate formation and neutrophil entrance to injured site

vascular response of inflammatory phase

- vasocontriction, coagulation


- chem mediators are released


- vasodilation later

3 stages of clot formation?

1. thromboplastin is formed


2. prothrombin is converted to thrombin due to interaction with thromboplastin


3. thrombin changes from soluble fibrinogen to insoluble fibrin coagulating into a network localizing the injury

What ends the inflammatory stage?

Fibrin clot: Leukocytes phagocytize the remaining debris

What causes chronic inflammation?

Occurs when acute inflammatory response does not eliminate injuring agent


- tissue not restored to normal physiologic state

How does the fibroblastic repair phase begin?

Commences when macrophages have finished


- cap buds begin to proliferate


- reactino to hypoxia


- create revascularization

What signals the beginning of stage 3?

tensile strength increases, fibroblastic activity slows

Maturation and remodeling is characterized by..?

Remodeling of scar tissue according to tensile forces


- tissue will gradually assume normal appearance

What should u do actively during the remodeling phase?

Controlled activity should be added


- work towards regaining normal flexibility and strength

Factors that impede healing?

- extent of injury


- edema


- hemorrhage


- poor vascular supply


- separation of tissue


- muscle spasm


- atrophy


- infection


- health, age, nutrition

Tell me about cartilage healing boi

- Limited capacity to heal


- little or no direct blood supply

Tell me about dat ligament healing my n*gga

similar to vascular tissues, 3 phases


- repair will involved random laying down of collagen which will mature and realign in reaction to joint stresses and strain

What types of factors affect the healing of ligaments?

- surgically repaired ligaments r stronger due to decreased scar formation


- exercised ligaments r stronger as opposed to immobilized

what is required for good tensile strength of tendon healing?

an abundance of collagen


- initially injured tendon will adhere to surrounding tissues

5 stages of acute fractures?

- hematoma formation


- cellular proliferation


- callus formation


- ossification


- remodeling

short term goals of injury rehab?

- control pain


- maintain/increase flexibility


- increase/restore strength


- maintain cardiorespiratory fitness

Long term goals of rehab?

return to practice/competition as quickly and as safely as possible

factors to consider when developing a RTP plan?

- Mechanism of injury


- age


- anatomical structures involved


- injury severity

Acute management goals?

Manage injury site - bleeding, pain, inflammation


- Prevent secondary complications


- support and stabilize injury


- implement a home program (RICE, basic ROM)

Effects of cold?

Vasoconstriction or vessels


- decrease blood flow, inflammation, cellular waste production

Hunting response?

bodys reaction ice when temp reaches 10 degrees


- vasodilation followed by vasoconstriction

fibroplastic repair phase goals?

control pain, reduce swelling


- scar is forming and pain is decreasing with active and passive ROM

Goals of maturation and remodeling?

- Return to activity


- collagen fibers need stresses and strains to align


- functional training


- strengthening exercises