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16 Cards in this Set
- Front
- Back
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Ant. Cerebral Artery |
Contra Leg-Foot sensory and motor loss |
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MCA |
Broca and Wernicke’s aphasia if dominant hemisphere, contra trunk-arm-face sensory +motor loss |
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Posterior Cerebral A |
Visual cortex, cortical blindness (eye works but cortex does not identify or see) |
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Ant. Communicating Artery |
Visual field defect due to supply to optic chiasm
Circle of Willis = AD Polycystic Kidney Dz, connective tissue defects like Ehlers-Danlos syn, Marfan’s. Also with age, smoking, HTN, smoking
Aneurysms of the anterior communicating artery are the most common circle of Willis aneurysm[1] and can cause visual field defects such as bitemporal hemianopsia (due to compression of the optic chiasm) |
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Posterior Communicating AA |
CN III palsy Berry Aneurysms, as above |
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CVA- Lateral Striate Div of MCA
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Internal capsular stroke, caudate, putamen, globus pallidus = pure, dense, motor paralysis contra to injury typical of capsular stroke These strokes are the common sequelae of the small-vessel disease seen in amyloidosis, DM, HTN assoc with Charcot-Bouchard Aneurysms and bleeds |
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CVA-Watershed Stroke
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“Hind Teat” strokes in zones between PCA and MCA and between Ant. Cerebral AA and MCA. Upper leg/upper arm weakness and visual processing defects |
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PICA
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Wallenberg’s syndrome: nystagmus ipsilateral ataxia, nausea, vomiting, Horner’s Syndrome
This syndrome is characterized by sensory deficits affecting the trunk (torso) and extremities on the opposite side of the infarction and sensory deficits affecting the face and cranial nerves on the same side with the infarct.
Specifically, there is a loss of pain and temperature sensation on the contralateral (opposite) side of the body and ipsilateral (same) side of the face. This crossed finding is diagnostic for the syndrome. Clinical symptoms include swallowing difficulty, or dysphagia,[1] slurred speech, ataxia, facial pain, vertigo, nystagmus, Horner's syndrome, diplopia, and possibly palatal myoclonus. |
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Basilar AA
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“Locked-in Syndrome” can hear and is aware but cannot move any skeletal muscles, can spare vs include the eyes, aka ventral pontine syndrome |
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Cerebellar Hemispheres
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Intention tremor, limb ataxia, falling toward side of lesion when unilateral Acoustic Neuroma |
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Cerebellar Vermis
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Truncal ataxia, dysarthria
Dysarthria is a motor speech disorder. It results from impaired movement of the muscles used for speech production, including the lips, tongue, vocal folds |
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Cavernous Venous Sinus Thrombosis |
CN III-VI palsies due to course within this vessel, retinal hemorrhage or papilledema, can also present similar to meningitis Assoc with exgtension of sinusitis or eye or dental infections with strep and staph |
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Right Parietal Lobe
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Spatial neglect of contralateral side |
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Mamillary Bodies
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Thiamine deficiency, wernicke-korsakoff syndrome = confusion, confabulation, opthalmoplegia, ataxia |
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Paramedian Pontine Reticular Formation (PPRF)
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Eye mvt coordination, unable to look toward side of lesion |
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PICA |
Nystagmus ipsilateral ataxia, nausea, vomiting, Horner’s Syndrome
Specifically, there is a loss of pain and temperature sensation on the contralateral (opposite) side of the body and ipsilateral (same) side of the face. This crossed finding is diagnostic for the syndrome. Clinical symptoms include swallowing difficulty, or dysphagia,[1] slurred speech, ataxia, facial pain, vertigo, nystagmus, Horner's syndrome, diplopia, and possibly palatal myoclonus. |
