CNS And Fatigue Analysis

Results The CNS has an important role in functions of the body with carrying messages to and from the brain and body. Without neuro-messages being transmitted, the skeletal muscles would be unable to move. Therefore, the brain and body need to communicate. “There can be little doubt that the regulation of central motor output is crucial in determining exercise performance” (Hargreaves, 2008, p. 1541). Although with the complexity of the CNS understanding what the role the CNS has to do with fatigue can be perplexing, considering all systems interact together during rest and exercise. Many researchers have discovered the complex nature of fatigue and how CNS and PNS interact leading to fatigue as well as prevent extreme exhaustion …show more content…
Early research did not consider nor have knowledge of buffering capacities within human blood and therefore, failed to acknowledge the strong ion differences that increase re-synthesis of lactate (Boning & Maassen, 2008; Boning, Strobel, Beneke, & Maassen, 2005; Kemp, 2005; Kemp et al., 2006). Even with knowledge of buffering capacity, educators and physiologists still seem to teach the old information about lactate production and acidosis. Educators need to teach lactate production and acidosis as two different events that happen in the body during exercise and not a cause and effect of events (Prakash, Robergs, Miller, Gladden, Jones, Stringer, Wasserman, Moll, Gros, Rowlands, Sahlin, & Beneke, 2008). Without lactate the body would fatigue quicker and exercise performance would severely be affected (Macedo, Lazarim, Silva, Tessuti, & Hohl, 2009; Robergs et al., 2004). Since lactate is not a producer of acidosis, researchers continue to predict that nonmitochondrial ATP turnover is a logical reason for acidosis created during exercise (Robergs et al., 2005). Education and research experts need a better direction to further develop and guide knowledge in lactate production and acidosis that occurs from …show more content…
Current research evidence has validated that lactate production does not lead to acidosis. In fact, nonmitochondrial ATP turnover during exercise disproves the “lactic acidosis” concept (Robergs et al., 2004). Muscle buffering capacity of protons, if evaluated correctly, might provide more evidence of the functions of lactate production and accumulated acidosis during exercise. Those concepts could lead to a greater understanding of fatigue. Educators and researchers are strongly advised to not continue teaching the errors found about lactate with cause and effect of acidosis and only teach more recent factual information. This knowledge will broaden the understanding of lactate performance during