Other papers presented positive immunoexpression of p53 protein, yet TP53 mutations seem to be uncommon events in pleomorphic adenomas (PA), adenoid cystic carcinomas (ACC), mucoepidermoid carcinomas (MEC) and carcinomas ex pleomorphic adenomas (CAexPA). Also, I will talk about other diseases produce from exposured TP53 to uranium (11). The most common mechanism is the loss of enzymes in the human body. The main two involved are Glurathione (GSH) and SuperOxide Dimutase
Other papers presented positive immunoexpression of p53 protein, yet TP53 mutations seem to be uncommon events in pleomorphic adenomas (PA), adenoid cystic carcinomas (ACC), mucoepidermoid carcinomas (MEC) and carcinomas ex pleomorphic adenomas (CAexPA). Also, I will talk about other diseases produce from exposured TP53 to uranium (11). The most common mechanism is the loss of enzymes in the human body. The main two involved are Glurathione (GSH) and SuperOxide Dimutase